Ocular toxoplasmosis is caused by the protozoa parasite Toxoplasma gondii, and can be acquired congenitally or by ingesting uncooked meat infected with cysts or vegetables and water contaminated by oocysts shed by cats [1]. T. gondii infects up to a third of the world’s population and is the most frequent etiology of infectious intraocular inflammation (uveitis) [2]. In some countries, up to 50% of all cases of posterior uveitis in a given population can be attributed to toxoplasmosis [3-4]. Toxoplasmic retinochoroiditis can be seen in the setting of congenital or postnatally acquired disease as a result of acute infection or recurrence [5-6]. This disease typically affects the posterior pole of one eye, and the lesions can be solitary, multiple or satellite to a pigmented retinal scar. The retina is the primary site of T. gondii infection in the eye but the choroid, vitreous and anterior chamber are also involved by inflammation. The choroid is secondarily affected, but choroidal lesions do not occur in the absence of retinal infection. An intense, secondary iridocyclitis may also be present [6-7]. The optic nerve head can also be involved in ocular toxoplasmosis [8]. Ocular complications include choroidal neovascularization, cataract, glaucoma, optic nerve atrophy and retinal detachment, more frequently in children [9]. The appearance of toxoplasmic retinochoroiditis lesions vary. Their duration and intensity may be related to host, parasite, or environmental factors. Genotyping of the infecting parasite appears to be an important determinant of disease severity in immunocompetent patients.⁷ Retinal vasculitis and associated inflammatory reactions may be the only ophthalmic sign during the early stages of a newly acquired T. gondii infection. Later development of retinitis or scars consistent with toxoplasmic retinochoroiditis in the same eye suggests that the initial, isolated inflammation may have been caused by the parasites [10]. Transplacental transmission of T. gondii to the fetus during pregnancy is another important source of infection. The mother can transmit toxoplasmosis to the fetus if infected by T. gondii during pregnancy or a few months before conception [11]. The infection can result in visual and hearing loss, mental and psychomotor retardation, seizures, hematological abnormalities, hepatosplenomegaly, or death [12]. Retinochoroidal scars are the most characteristic eye manifestation of a congenital or prenatal infection [13].