Toxocariasis is a cosmopolitan affection linked to the tissue presence of ascarid larvae evolving naturally in dogs or cats. These larvae are in a parasitic impasse in humans. Toxocara canis, a dog nematode, is the most frequently involved. It may also be Toxocara cati, a cat nematode, or more rarely, wild animal larvae causing clinically similar syndromes. The definitive host of this parasite is the cat or the dog. The adult form of the parasite lives in the small intestine of these animals. Contamination of animals can occur by ingestion of the viable form of the parasite, its embryonic form or even an infection in utero (trans-placental) [4]. In dogs, contamination can occur by ingesting embryonated eggs present in food soiled by parasitized excrement resulting in the formation of mature adults in the lumen of the small intestine in 3 to 4 weeks. The eggs produced by these adults, up to 200,000 per day, will be disseminated by the feces of the animals in the external environment, where they will become infective. Human infestation starts from the digestive tract towards the portal vein, then the larvae migrate into the pulmonary veins and are disseminated in the tissues, in particular the liver, lungs, muscles, eyes and nerves [5]. Eosinophilic granulomas are formed around the larva and then an antigenic inflammatory reaction occurs. Humans become contaminated by absorbing embryonated eggs or larvae [3], humans can, in turn, be contaminated by the faecal-oral route. Children are also more exposed to this disease due to more contact with the outside environment and more frequent hand-to-mouth carriage. Reviewing the literature, there are two forms of toxocariasis in adults, the asymptomatic form resulting from a minimal infestation, the diagnosis of which is made on the occasion of hypereosinophilia, and the minor form due to a moderate infestation, associating non-specific signs: abdominal pain, persistent asthenia, rheumatological, pulmonary and skin involvement [4, 5] Ocular, cardiac and neurological manifestations have been described [6]. Definitive diagnosis is based on serology. The techniques used are mainly of the immunoenzymatic type (Elisa) with a sensitivity estimated at 90%. Their specificity is not perfect because there are often cross-reacts with other parasites such as nematodes, especially in patients residing in tropical areas. The Western blot technique can detect IgG directed against specific antigens and therefore confirm the results obtained by ELISA [7]. In our study, toxocariasis was discovered during recent hypereosinophilia in 7 cases. We found the notion of dog breeding in the majority of cases. The notion of consumption of forest herbs during several hikes and the presence of pet dogs and cats from a young age was revealed during the interrogation in the case of pulmonary toxocariasis and hepatic. The diagnosis was confirmed by serology, which was positive by the ELISA technique and Western blot [8]. In our country, the prevalence of ocular toxocarosis is not well known: Clinically, this parasitosis can manifest itself by a granuloma of the posterior pole, a peripheral granuloma, chronic endophthalmitis, by posterior uveitis or panuveitis. It represents a rare etiology of posterior uveitis in adults. There is a predilection for young adults because of their frequent contact with animals. It is most often a unilateral posterior uveitis, but bilateral forms have been described [9], with the presence of a deep granuloma of the posterior pole or the periphery. In adults, the focus of chorioretinitis is often more peripheral, typically associated with significant vitreoretinal traction which can be complicated by retinal detachment [10, 11].