The most common cause of diarrhea is infectious etiologies. While the most common infection is viral gastroenteritis, bacteria such as Campylobacter, Salmonella, and Shigella are the most common cause of acute diarrhea [1]. One of the leading pathogens that cause gastroenteritis in developed countries is the Campylobacter species. Campylobacteriosis has early and late complications that can be deleterious. For instant, urticaria, erythema nodosum, cholecystitis, peritonitis, and abdominal aortic septic pseudoaneurysm. The late course of the disease presents as reactive arthritis and Guillain-Barré syndrome. Campylobacter infection may progress to bacteremia in immunocompetent and immunocompromised individuals (2). Pericarditis and myocarditis can be associated with a late complication of campylobacter gastroenteritis, as demonstrated in our patient [3]. The development of cardiac manifestations presents most often involves young adults or teenagers. As in most gastroenteritis illnesses, symptoms begin with fevers, abdominal pain, and diarrhea, after which symptoms of concurrent chest pain, discomfort, or tightness ensue within 48-72 hours [5]. Therefore, a reported history of chest discomfort following bouts of diarrhea should prompt physicians to include Campylobacteriosis in their differential [4]. The pathophysiology of perimyocarditis secondary to Campylobacter gastroenteritis is not fully understood. Still, the combination of the number of organisms consumed, the virulence of the infecting strain, and the host immunity are all crucial factors in the patient’s clinical presentation. Several factors are beginning to be just understood, and two proposed models have been reviewed in the literature. One model suggested that the pathogen or toxin-induced may be responsible for the acute presentation of the carditis within 48-72 hours. The alternative model offered that the immunological response such as systemic circulation of immune complexes or cytotoxic T Cell attack presents with the later presentation of the cardiac embodiments [6-9].

Regardless of the proposed pathogenies of the disease, almost all the immunocompetent patients with gastroenteritis secondary due to campylobacter infection in the literature have not been reported to experience bacteremia with the disease. However, this doesn’t exclude the likelihood of the pathogen harboring transient bacteremia as its mechanism of action [10]. Lastly, because most Campylobacter infections are self-limited and have limited efficacy of antimicrobials, antibiotic therapy is only recommended for individuals with severe disease. For instance, patients with bloody stools, a high temperature, an extraintestinal infection such as myocarditis, pericarditis or perimyocarditis, increasing or recurrent symptoms, or symptoms that persist for more than a week are considered to have a severe illness [11].